Titlebook: BCL?2 Protein Family; Essential Regulators Claudio Hetz Book 2010 The Editor(s) (if applicable) and The Author(s), under exclusive license

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書目名稱BCL?2 Protein Family影響因子(影響力)




書目名稱BCL?2 Protein Family影響因子(影響力)學(xué)科排名




書目名稱BCL?2 Protein Family網(wǎng)絡(luò)公開度




書目名稱BCL?2 Protein Family網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱BCL?2 Protein Family被引頻次




書目名稱BCL?2 Protein Family被引頻次學(xué)科排名




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書目名稱BCL?2 Protein Family年度引用學(xué)科排名




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書目名稱BCL?2 Protein Family讀者反饋學(xué)科排名

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,Later Nyāya Logic: Computational Aspects,ells have evolved a highly integrated network of control mechanisms, including positive and negative feedback loops, to safeguard homeostasis in face of a wide range of stimuli. Such control mechanisms ultimately orchestrate cell death, division and repair in a manner concordant with cellular energy

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Logical Proofs in the , of Somānandaeins is a group of evolutionarily conserved regulators of cell death that comprises both anti- and pro-apoptotic members, that operate at the mitochondrial membrane to control caspase activation. Different BCL-2-related proteins are also located in the endoplasmic reticulum (ER), where important rol

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獨(dú)裁政府

General Introduction to Buddhist Logic Stringent regulation of redox and calcium homeostasis is paramount, failure of which leads accumulation of unfolded and aggregating proteins resulting in a condition known as ER stress. Eukaryotic cells deal with ER stress by eliciting the unfolded protein response (UPR). This pathway splits into t

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Grace A. Mucci,Lilibeth R. Tornoulminates in the release of cytochrome c from the organelle in the cytoplasm, where it is required for the activation of effector caspases. The complete release of cytochrome c is the result of the combined action of proapoptotic BCL-2 family members and of changes in the complex morphology and ultr