Titlebook: Atherosclerosis — is it reversible?; Gotthard Schettler,Eduard Friedrich Stange,Robert Conference proceedings 1978 Springer-Verlag Berlin

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https://doi.org/10.1007/978-94-6265-162-3intracellular transport and secretion of chylomicrons and VLDL were pointed out previously (12). It seems pertinent to point out that the VLDL and nascent chylomicrons secreted by the intestine do not have their full complement of apoproteins (23) and acquire them in the circulation.

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The Porcine Species, an Useful Model in Comparative and Experimental Arteriosclerosis Researchadded to the food have an influence on the development of atherosclerosis in swine. The natural occurrence of atherosclerosis can be accelerated by fat diets. Unsaturated fat diets result in more retardation than the use of saturated fats.

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Mechanisms of Lipid Deposition in the Artery Wallified from an article published recently (8) and based, in part, on CHEN’s work which was done in our laboratory (5). These diagrams show endothelial cells, the internal elastica, a few media cells, and elastic membranes.

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Monoclonal Character of Human Atherosclerotic Plaquesulations, their embryogenesis and turnover as it reates to age, “wear and tear” in artery walls, and atherosclerosis. Information can be gained on some points by animal experiments with isotopic tracers. Furthermore, insight into the population dynamics of human tissue cells is obtained by using an

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