Titlebook: Apoptosis; Enrico Mihich,Robert T. Schimke Book 1994 Springer Science+Business Media New York 1994 Antigen.DNA.apoptosis.cancer.cell.cell

INFER

書目名稱Apoptosis影響因子(影響力)




書目名稱Apoptosis影響因子(影響力)學(xué)科排名




書目名稱Apoptosis網(wǎng)絡(luò)公開度




書目名稱Apoptosis網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Apoptosis被引頻次




書目名稱Apoptosis被引頻次學(xué)科排名




書目名稱Apoptosis年度引用




書目名稱Apoptosis年度引用學(xué)科排名




書目名稱Apoptosis讀者反饋




書目名稱Apoptosis讀者反饋學(xué)科排名

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Elemente der Achten Nebengruppedependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for developing factor-independent growth. In the absence of IL-3, 32D cells rapidly arrest in the Gl phase of the cell cycle and eventually undergo apoptosis..

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Regulation of Apoptosis in Interleukin-3-Dependent Myeloid Progenitor Cells,dependent 32Dcl3 murine myeloid cell line. 32D cells are absolutely dependent upon IL-3 for proliferation and survival,. and have no propensity for developing factor-independent growth. In the absence of IL-3, 32D cells rapidly arrest in the Gl phase of the cell cycle and eventually undergo apoptosis..

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Wild Type p53 Activity Contributes to Dependence on Hematopoietic Survival Factors,s found in cancer cells are typically devoid of such activities. Hence, the principal outcome of such mutations is probably the inactivation of the tumor suppressor function of wt p53. In addition, at least certain mutations may also confer a distinct gain of function., thereby possibly contributing a novel oncogenic activity to the affected cell.