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Titlebook: Angiotensin and the Heart; H. Grobecker,Gerd Heusch,B. E. Strauer Book 1993 Dr. Dietrich Steinkopff Verlag GmbH & Co. KG, Darmstadt 1993 A

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11#
發(fā)表于 2025-3-23 13:01:43 | 只看該作者
https://doi.org/10.1007/978-3-030-69583-5oint to a role in long-term cardiovascular regulation for endogenous RAS in target tissues. This article focuses on the multiple effects of tissue angiotensin enzyme (ACE) and angiotensin II (Ang II), its active peptide product. Ang II has been shown to be a potent growth factor in vascular smooth m
12#
發(fā)表于 2025-3-23 15:36:11 | 只看該作者
13#
發(fā)表于 2025-3-23 19:44:59 | 只看該作者
14#
發(fā)表于 2025-3-23 23:39:17 | 只看該作者
https://doi.org/10.1007/978-3-030-54001-2tors have also been used in the treatment of patients with coronary artery disease (CAD), since from experimental data an antiischemic action of these agents is suggested. Antiischemic effects of ACE-inhibitors may be exerted through a reduction of myocardial oxygen demand, by a reduction of angiote
15#
發(fā)表于 2025-3-24 03:37:23 | 只看該作者
16#
發(fā)表于 2025-3-24 06:41:20 | 只看該作者
17#
發(fā)表于 2025-3-24 12:14:53 | 只看該作者
EU Islands and the Clean Energy Transition patients with arterial hypertension often have symptoms of angina pectoris and a positive exercise tolerance test. The angina pectoris-symptoms in patients with arterial hypertension are due to functional and structural alterations of the coronary microcirculation. Consequently, an antihypertensive
18#
發(fā)表于 2025-3-24 15:27:34 | 只看該作者
19#
發(fā)表于 2025-3-24 21:08:44 | 只看該作者
The Policy Paradigm from 1997 to 2007,ocardium have been summarized under the term “l(fā)eft ventricular remodeling” post myocardial infarction. An increase in left ventricular volume may be a consequence, and associated with an adverse prognosis. Although left ventricular dilatation may increase stroke volume and, thus, be compensatory at
20#
發(fā)表于 2025-3-25 00:20:35 | 只看該作者
The Policy Paradigm from 1997 to 2007,nd positive inotropic agent. This has raised the possibility that angiotensin converting enzyme (ACE) inhibitors might be “cardioprotective” (that is, might attenuate myocardial injury, dysfunction and necrosis) in the setting of acute ischemia and infarction. Captopril, enalapril and ramipril have,
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