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Titlebook: Angiogenesis in Health, Disease and Malignancy; Ali Salajegheh Book 2016 Springer International Publishing Switzerland 2016 Gene function.

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發(fā)表于 2025-3-27 00:33:54 | 只看該作者
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發(fā)表于 2025-3-27 01:55:17 | 只看該作者
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發(fā)表于 2025-3-27 06:06:54 | 只看該作者
Vassilios Karakostas,Dennis Dieksrs to disease and malignancy, cancer proliferation and progression will be discussed. New insights into the role of angiogenesis and the therapeutic inhibition of its regulators will be investigated due to the great potential for exploitation in the development of a novel treatment for cancer.
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發(fā)表于 2025-3-27 13:25:39 | 只看該作者
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發(fā)表于 2025-3-27 15:09:55 | 只看該作者
Vassilios Karakostas,Dennis Dieksas a neuroprotective role. Cancer viability and spread is also supported by the expression of ANG in many malignancies. Angiogenin drives tumour angiogenesis, promote cancer growth and metastasis by induction of ribosomal RNA transcription that enables angiogenesis initiation by VEGF and other vascu
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發(fā)表于 2025-3-28 01:28:28 | 只看該作者
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發(fā)表于 2025-3-28 04:15:16 | 只看該作者
European Studies in Philosophy of Scienceochondrial electron transport chain in aerobic respiration, and hence it can be concluded that hypoxia-regulated miR-210 triggers the metabolic switch from aerobic to anaerobic respiration in cancer cells via downregulation of COX10. This, in turn, promotes the survival of tumour cells under hypoxic
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發(fā)表于 2025-3-28 07:31:07 | 只看該作者
Agota Szende,Mark Oppe,Frank de Charroexert its functions and promote adhesion of endothelial cells. In rheumatoid arthritis CYR61 was found to play a critical role in proliferation in fibroblast-like synoviocytes (FLS) and also likely to contribute to the hyperplasia of synovial lining cells and eventuating in joint destruction. CYR61
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發(fā)表于 2025-3-28 11:00:45 | 只看該作者
Guidance to users of EQ-5D value sets,ent of new capillaries in tumours directly relate to the release of angiogenic factors related to EGF and its multitude of receptors. As such, the intensity of neovascularisation directly correlates with the development of metastasis and thus the number of EGF, EGFR and VEGF. Therefore, various curr
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