Titlebook: Aging at the Molecular Level; Thomas Zglinicki (Professor of Cellular Gerontolog Book 2003 Springer Science+Business Media Dordrecht 2003

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978-90-481-6482-0Springer Science+Business Media Dordrecht 2003

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Oxidative Damage, Somatic Mutations and Cellular Aging,Many theories have been proposed to explain the aging process. One of the more widely accepted of these is the free radical theory of aging. First presented in 1954, it postulates that aging is caused by the accumulation of damage to the macromolecules of somatic cells by free radicals [1].

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John Connell,Phil McManus,Xuesong Dingolecular damage, and the limited ability of cells, tissues, organs, and individuals to repair it or to maintain function despite it, is the principal driving force of the aging process. A major cause for molecular damage, although by far not the only one (see Chapters 11, 12 and 13, this volume), ar

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John Connell,Phil McManus,Xuesong Ding aging in various biological systems. All aerobic organisms are faced with a load of unwanted side reactions caused by the incomplete reduction of oxygen, for example during electron transport and oxidative phosphorylation. Major oxygen radical species are formed such as superoxide and hydroxyl radi

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Chirurgie des Sinus pilonidalis appear much earlier than in the average normal individual; thus, the premature aging disorders are useful models for the study of the aging process (Figure 1). Werner syndrome (WS) is the most characterized premature aging disorder. Patients with WS have a large number of signs and symptoms of norm