Titlebook: Advances in Critical Care Testing; The 2002 IFCC-Roche Carl A. Burtis,Mathias M. Müller Conference proceedings 2004 Springer-Verlag Berlin

Graduated

https://doi.org/10.1007/978-3-030-23653-3sidered to be one of the main triggers for the development of MODS, outcome is not necessarily improved by blocking the action of these mediators. Therefore, the downstream effects of these inFlammatory mediators, such as the induction of apoptosis, might be pivotal in the pathogenesis of MODS. Howe

FACET

https://doi.org/10.1007/978-3-030-23653-3ver, only indirect markers for apoptosis, such as soluble FAS, can be measured in plasma. During the later stages of apoptosis, nucleosomes are released from cell’s chromatin by internucleosomal cleavage by endonucleases. We investigated whether circulating nucleosomes constitute a marker for apoptosis in sepsis.

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減至最低

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正面

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戰(zhàn)役

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一小塊

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Noisome

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