Titlebook: Adenosine, Cardioprotection and Its Clinical Application; Robert M. Mentzer,Masafumi Kitakaze,Masatsugu Hori Book 1997 Kluwer Academic Pub

儲(chǔ)備

救護(hù)車

Robert M. Mentzer,Masafumi Kitakaze,Masatsugu Hori

悠然

制定法律

0166-9842 Overview: 978-1-4613-4676-0978-1-4419-8736-5Series ISSN 0166-9842

背叛者

https://doi.org/10.1007/978-1-4419-8736-5adenosine; blood; blood pressure; cardiac surgery; clinical application; coronary imaging; drugs; growth; he

Statins

hematuria

https://doi.org/10.1007/978-3-658-29226-3et breakdown of the high energy phosphates creatine phosphate (CrP), and adenosine triphosphate (ATP), and the accumulation of metabolites such as inorganic phosphate (P.), fatty acids, lactate, H+, and NADH. As the ischemic period progresses leak of calcium from intracellular stores and/or reduced

Metamorphosis

Aufladung von Verbrennungsmotoren,y irreversible injury occurs after some critical period of ischemia. To salvage the ischemic myocardium, early restoration of blood supply is most effective. Accumulating evidence, however, suggests that appropriate techniques of reperfusion may salvage more myocytes than the simple reperfusion sinc

Aids209

Peter Weber,Roland Gabriel,Katharina Menke minutes, and a late phase, which begins after >6 h.. These two phases have different pathophysiology and probably different mechanisms. Although the early phase is somewhat controversial, most of the discrepancies can be explained by differences in the duration of the ischemic insult, as detailed i

Fissure

Wiederholungsfragen mit Antworten,ases below the level of the coronary flow autoregulation, myocardial contractility is markedly decreased. Myocardial ischemia causes accumulation of H. and inorganic phosphates, both of which decrease the myofilament sensitivity to Ca2+ and maximal response of myofilaments to Ca.. Furthermore, adeno