Titlebook: Acute Leukemias VIII; Prognostic Factors a T. Büchner,W. Hiddemann,U. Creutzig Conference proceedings 2001 Springer-Verlag Berlin Heidelber

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https://doi.org/10.1007/978-3-642-18987-6ocations is underscored by the use of these translocations in developing risk-directed therapy for many forms of acute leukemia. However, the demonstration that the translocations can be causal events for leukemia immediately raises an important, related question regarding the pathogenesis of leukem

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傳授知識

Avatare und die Usability von Websitessome 7 harbours tumor suppressor genes which play a role in the pathogenesis of these malignancies (Atkin et al. 1993; Zenklusen et al. 1996). In particular, in myeloid disorders loss of -7/7q- are among the most frequent chromosome abnormalities. These aberrations are associated with myelodysplasti

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季雨

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Computer Supported Cooperative Workleukemia (APL). According to the majority of reports the translocation t(15;17)(q22;ql2–21) is detected in 70% to 100% of cases [.]. PML/RARa fusion transcript from add(15q) is beleived to play crucial role in leukemogenesis of this type of leukemia [.]. It’s presence remains the predictor of favour

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Jeremy N. Bailenson,Andrew C. BeallDS-AML), in relation to hematologic and clinical status of patients. Cytogenetic analyses were performed in 85 untreated patients; 64 with primary, and 21 with secondary AML. The studies were carried out on at least 20 GTG-banded mitoses, obtained after 24-hour unstimulated culture of bone marrow ce

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Michael Schwarzbach-Dobson,Franziska Wenzelthe study was to determine the influence of erythroid and/or megakaryocytic dysplasia (EMD) in diagnostic bone marrow smears of patients under 65 years with de novo AML and a normal karyotype on their response to therapy and prognosis. EMD was diagnosed when more than 25% erythroblasts and/or 50% me